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When the major site of phosphorylation of DOR is mutated (S363A), it is possible to observe a deltorphin I-induced endocytosis (Navratilova et al., Concerning the rKOR, the phosphorylation-defective mutant S369E is unable to internalize upon U50,488 exposure demonstrating the role of receptor phosphorylation in endocytosis (Mclaughlin et al., While those data indicate that MOR and DOR phosphorylation would favor their endocytosis, KOR phosphorylation would be essential to promote its internalization

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