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Research involving reproductive-aged men demonstrated that urinary levels of phthalate monoesters and oxidative metabolites were significantly associated with increased sperm DNA damage, indicating direct genotoxic effects of these ubiquitous chemicals ( 4.1.1.3 Hormonal regulation and EED impact The fundamental role of androgens in spermatogenesis regulation provides a critical framework for understanding EED reproductive toxicity
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The analysis of the population reveals a notable progressive increase of the dosage of BTX-A over time, a trend that has been previously documented by other researchers [27]
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