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Reactive astrogliosis in neuroinfection is thus defensive and neuroprotective: inhibition of astrocytic reactivity, for example by knocking out GFAP, 231 or by genetic deletion of NK-1R receptor for substance P, which suppresses reactive astrogliosis that defines resistance to N

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Nowadays, although some HDACi including VPA, lamotrigine and SAHA [172] are approved or enrolled in clinical trials for the treatment of epilepsy, they always show some adverse effects such as hepatic injury, teratogenicity and so on, which are largely attributed to the global inhibition of HDACs for these drugs