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Tumor necrosis factor-, interleukin-1, and interferon- stimulate -secretase-mediated cleavage of amyloid precursor protein through a JNK-dependent MAPK pathway
Dla form ARG sytuacja jest bardziej zoona
doi: 10.1158/1078-0432.CCR-24-3419 175 PopoviVBuriMMihailoviAAimi RemikoviMVukeljiNBatrieviMet al
Future research should build on this framework by prioritizing well-powered, longitudinal, and mechanistically integrated study designs that combine precise cycle phase verification with multi-omics approaches (e.g., metabolomics, transcriptomics, and microbiome profiling)
