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Notably, as a result of (skewed) X chromosome inactivation, heterozygous females may be susceptible to the same pathophysiologic phenotype as their hemizygous male counterparts.30,93,156 G6PD-deficient neonates who have coinherited a mutation in the uridine-diphosphate-glucuronosyl-transferase-1 (UGT1A1) promoter are particularly at risk for developing neonatal jaundice and even kernicterus.94,205 A small proportion of G6PD-deficient individuals display the phenotype of chronic nonspherocytic hemolytic anemia

M., and Ribeiro M

By contrast, at lower doses (1080 g/kg, i.p.), all tested neurons in the mediodorsal nucleus (MD) of the thalamus increased both their firing frequency and bursting, while in the reticular nucleus, half of the neurons were excited and the other half inhibited