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developed glycosylated triple-interaction stabilized polymeric siRNA NPs (Gal-NP@siRNA) to target -site amyloid precursor protein cleaving enzyme 1 (BACE1) in an amyloid precursor protein (APP)/PS1 transgenic AD mouse model [68]

Metabolomics analyses revealed a time-dependent increase in tricarboxylic acid cycle intermediates during ES treatment, suggesting that copper toxicity primarily disrupts the tricarboxylic acid cycle rather than oxidative phosphorylation (180,181)

Discussion In previous studies using this NEC model, we and others 18,19 demonstrated features of NEC with severe bowel damage in pups, inflammatory changes, and increased oxidative stress accompanied by offspring brain injury

Our findings were comparable to those of Saoudi and his colleagues (2021) 2 in terms of the phytochemical makeup