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Dysfunction of cytotoxic T lymphocyte induced by hepatoma cells through the Gln-GLS2-Endoplasmic reticulum stress pathway
Hyperglycemic stimulation of PKC- in atrial cardiomyocytes activates the NF-B/TGF- signaling pathway, leading to increased expression of pro-inflammatory and pro-fibrotic mediators such as TNF-, TGF-, and signaling molecules like p-IB, p38 MAPK, Cav1.2, and NCX
93 Animal studies have indicated that age-related disruption of nave T cell survival and homeostasis depends on alterations in the secondary lymphoid environment and IL-7 (a maintenance factor) signaling
D., Wood, D., Reissner, K
