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B., Chen C

These results may explain, in part, the finding that low-dose dietary zinc supplementation extended the lifespan of SOD1 G39A transgenic mice (Ermilova et al., 2005), as this mutation is known to reduce Sod1s affinity for zinc (Crow et al., 1997)
The present results showed that the AR activity, which is considered as key enzyme of polyol pathway, was highly elevated in diabetic subjects ( Table 3 )

It is not clear whether glucose metabolism is the cause or the result of PD
