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Both exhibited lower hepatic GSH levels and GCL activity, which were prevented with hormone replacement [64]
Inhibiting IDH1 reduces damage and apoptosis in Ox-LDL-treated macrophages while increasing Nrf2 levels, thereby decreasing foam cell formation (Li B
& Guidi, A
A review on the possible molecular mechanism of action of N-acetylcysteine against insulin resistance and type-2 diabetes development
