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Since then, Siess definition has been adapted to be more comprehensive an imbalance between oxidants and antioxidants in favor of the oxidants, leading to a disruption of redox signaling and control, and/or molecular damage [7,19,22]. It is essential to appreciate this definition, given the complex cellular redox dynamics that occur in vivo

A study proved that p53 could bind to dipeptidyl-peptidase 4 (DPP4) to reduce the DPP4-dependent oxidization of PUFAs to inhibit ferroptosis in colorectal cancer cells[46]

Toll-like receptor 3 on adult human astrocytes triggers production of neuroprotective mediators