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It is known that JNK activation is mediated by oxidative stress and plays pathogenic roles in a diverse array of cellular programmes, including cell differentiation, movement, proliferation and death 34

Although the C-terminal SAM-binding site is located away from the catalytic center, it was revealed that SAM binding induces a structural rearrangement of the catalytic core that relieves autoinhibition and stabilizes an activated conformation (Prudova et al., 2006)

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Characterization of the differentiated antioxidant profile of human saliva