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Functional dystonia (FD) may resemble other forms of dystonia (OD) but has a different pathophysiology ( Historical Context Even after over a century of examination and consideration of its presentation, symptoms, and diagnostic criteria, FD remains a challenging movement disorder for neurologists to diagnose, and patients to understand ( One of the founders of neurology, Jean-Martin Charcot's description of hysteria mirrored the typical presentation of dystonia that doctors faced in the clinic decades later ( Through the mid-twentieth century, dystonia presentations such as blepharospasm were treated primarily with long-term psychotherapy and other, sometimes experimental psychiatric treatments ( By the 1970s, David Marsden popularized dystonia's neurological features and sidelined FD as a markedly uncommon entity ( Later, neurological cases of psychogenic dystonia responding to psychotherapeutic treatment were reported in the late 1970's and early 1980's ( Despite the clarification brought by initial and subsequently updated diagnostic criteria ( Risk Factors for Functional Dystonia FD has several predisposing vulnerabilities, some of which are similar to those of primary OD

10.1111/j.1469-7580.2009.01199.x [DOI] [PMC free article] [PubMed] [Google Scholar] Kang E

Therefore, although both pathways involve System Xc and cystine metabolism, there are essential differences in their downstream mechanisms, key effector molecules (such as GPX4 in ferroptosis and actin in disulfidptosis), and modes of cell death