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Annals of the New York Academy of Sciences, 1033(1), 117131

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Systemic and cellular defense strategies to cope with extracellular Hb and its oxidation products might not be efficient or sufficient enough to control the deleterious effects of these molecules deep inside the atherosclerotic plaque, the death zone. Comprehensive understanding the role of neovascularization, IPH and Hb release and oxidation on atherogenesis may lead to the development of novel therapeutics intended to interrupt these pathological events