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The accumulation of A leads to the overactivation of microglia and astrocytes, triggering release of pro-inflammatory cytokines, such as IL-1 and TNF-, that sustain chronic neuroinflammation [249]

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All in all, the different pathways involved in GSH homeostasis are an indication of the complex dynamic and quick turn over of this tripeptide, and provide clues for potential targets for a GSH-depleting, ferroptosis-inducing strategy