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Histone deacetylases including SIRT1, interact with STAT3 and negatively regulate its transcriptional activity by deacetylating critical lysine residues ( Short peptides are distinctive biomolecules that combine the benefits of traditional small molecules and mature proteins, offering new, more effective therapeutic agents with fewer side effects ( 2+ affinity comparable to that of albumins copper transport sites, forming an in vivo complex, glycyl-L-histidyl-L-lysine-Cu 2+ (GHK-Cu), which enhances its bioavailability (Figure 1A

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