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Mitochondria and Mitochondrial Cascades in Alzheimers Disease
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Additionally, MSI-induced frameshift mutations in TGFBR2 disrupt TGF- signaling, which normally functions as a tumor suppressor by regulating cell growth and differentiation
Lipid metabolism is generally dysregulated in TNBC, and fatty acid desaturases 1 and 2 (FADS1/2), which are responsible for PUFA biosynthesis, are highly expressed in TNBC with poor prognosis
