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[54] to interact with NCP1-L1 and to control its movement from the endocytic recycling compartment to plasma membranes in a cholesterol-dependent manner
Any suggestion to the contrary is misleading. Black, who left UKA last year, told the Guardian he had nothing to say at this point while Fudge also declined to comment
[31] A 2013 review article reported additional but weaker links to the loci of the genes TNFSF4 (rs7553711), Cathepsin H (rs34593439), and P2RY11-DNMT1 (rs2305795)
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