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Description
Another significant fate of tyrosine is conversion to the catecholamines
However, the molecular mechanisms responsible for the pathogenesis of COPD remain poorly understood
In conditions of acetaminophen toxicity the administration of N-acetylcysteine (NAC) has been shown to replenish GSH stores, to scavenge reactive oxygen species (ROS) in the mitochondria, and to enhance the metabolism of acetaminophen via the sulfation pathway
In the future, it will be particularly interesting to test our method with integral membrane proteins
