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These substances are often considered as risk factors for the development of cancer and thus act as indicators of endogenous oxidative DNA damage [46]
Lee and colleagues demonstrated that MOTS-C administration prevents diet-induced obesity and insulin resistance in mouse models (Lee et al., Cell Metabolism 2015
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Yao, Activation of the Nrf2-Keap 1 pathway in short-term iodide excess in thyroid in rats
