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In HT, the vitamin D-induced prevention of dendritic cells- dependent T-cell activation and inflammatory cytokines production and the restoration of the anti-inflammatory state through a restoration of the Th17/Treg ratio has been claimed as one of the beneficial mechanisms (68, 69)
However, we could not exclude the possibility that other alternative pathways might be involved in this inhibition

Th2 cytokines impair innate immune responses to rhinovirus in respiratory epithelial cells

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