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he site of redox modification on vulnerable molecules may undergo oxidative damage, resulting in an irreversible inhibitory action on proteins involved in synaptic transmission, as well as promoting mitochondrial dysfunction and excitotoxicity [294]

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Li, L., Stoeckert, C

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Following engraftment of SCXC-5052, mice were treated with vehicle, AraC (50 mg/kg/day intraperitoneally on days 15) as routinely performed in the laboratory [6], rosuvastatin (1 mg/kg/day by oral gavage on days 17, the maximum dose tolerated in patients [49]) or the combination (schema in Fig

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