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The primary underlying pathomechanism is most likely a decreased hepatic GHR expression related to insulin deficiency, resulting in GH-resistance and impaired hepatic IGF-1 synthesis, which stimulates pituitary GH-secretion by a feedback mechanism [188, 189]
J Braz J Nephrol 41:124130 Macedo E, Mehta RL (2009) Prerenal Failure: from Old Concepts to New Paradigms
The coordinated adaptation of the lipidome and proteome to suppress plaque development suggests synergistic molecular mechanisms that prevent vascular inflammation and lipid peroxidation, contributing to a highly effective strategy for cardioprotection in cetaceans [50]
