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Tyrosine hydroxylase is inactivated by catechol-quinones and converted to a redox-cycling quinoprotein: possible relevance to Parkinsons disease
Oxidative-nitrosative stress as a contributing factor to cardiovascular disease in subjects with diabetes
Cilostazol inhibits hyperglucose-induced vascular smooth muscle cell dysfunction by modulating the RAGE/ERK/NF-B signaling pathways
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