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[DOI] [PMC free article] [PubMed] [Google Scholar] 149.Fatemeh Y., Siassi F., Rahimi A., Koohdani F., Doostan F., Qorbani M., Sotoudeh G

In cancers, such as esophageal, prostate and cervical cancers, genes such as cyclin dependent kinase inhibitor 2A (CDKN2A, also known as p16INK4a), O-6-methylguanine-DNA methyltransferase (MGMT), retinoic acid receptor (RAR), PC3 and suppressor of cytokine signaling 1 (SOCS1), which were previously hypermethylated, have been demethylated through the action of natural compounds, such as genistein and capsaicin (9)

In support of this concept, excess intracellular hydrogen peroxide has been shown to regulate differentially the expression of various NFB component proteins

Changes in glutathione status and the antioxidant system in blood and in cancer cells associate with tumour growth in vivo