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glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells Mammalian copper homeostasis: physiological roles

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glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells Mammalian copper homeostasis: physiological roles

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glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells Mammalian copper homeostasis: physiological roles

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glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells Mammalian copper homeostasis: physiological roles

P.Mc EwenA

glutathione in breast milk copper excess induces autophagy dysfunction and mitochondrial ROS-ferroptosis progression, inhibits cellular biosynthesis of protein and lipid bovine mammary epithelial cells Mammalian copper homeostasis: physiological roles

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