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(2015) investigated BPC-157s interaction with the nitric oxide system in intestinal anastomosis models

--- The Human Study: What They Did The study followed patients diagnosed with chronic knee pain including osteoarthritis, patellofemoral syndrome, and post-injury degenerative changes who had not achieved adequate relief from standard interventions including physical therapy, NSAIDs, and corticosteroid injections

Given the striking decrease of 55% of cells entering the S-phase of the cell cycle upon treatment with 2 ng/mL and 10 ng/mL of BPC 157 and decreased ERK phosphorylation, it was suggested that BPC 157 acts as an antimitogenic agent by preventing ERK phosphorylation and the propagation of the mitogenic signal via the MAPK signaling cascade, triggered by VEGF [95]