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Although its precise cause remains unclear, recent evidence suggests that metabolic disturbances, particularly oxidative stress and mitochondrial dysfunction, may play a significant role in the pathogenesis of the disease
Following SE, mGluR5 activation has also been shown to enhance astrocytic calcium signals during the latency period of epileptogenesis in the pilocarpine model of TLE (45)
Emerging evidence also highlights the pleiotropic antioxidant effects of several antidiabetic agents
Effect of donepezil and tacrine on oxidative stress in intracerebral streptozotocin-induced model of dementia in mice
