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The continuous disruption leads to kelpids and hypertrophic scars which can be viewed as an exuberant wound healing response, explained by a primary barrier abnormality [189]

Animal studies revealed that the expression of GSH, SOD, GPX4, and SLC7A11 was significantly downregulated in the cerebrospinal fluid of EAE mice induced by MOG35-55 peptide injection

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177,178 In conclusion, mitochondria activate NLRP3 inflammasome and initiate inflammatory response through diversified mechanisms including mtDNA leakage, ROS production, ATP, mitophagy and interaction with lysosomes