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This is advantageous, since a significant proportion of H 2 O 2 generated in the ER during proinsulin folding is consumed again for PDI re-oxidation (Hudson et al

The induction of diabetes mellitus significantly elevated blood pressure, HbA1c, BUN, creatinine, and renal tissue levels of MDA, whereas SOD, CAT and GPx activities were significantly reduced

Furthermore, a second mechanism involved would be related to TGF--dependent apoptotic processes, producing an increase in caspase 8, which in turn acts as an inducer of caspases 3, 6 and 7, giving rise to apoptosis [29,46]

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