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Description
This phenotype is likely to be secondary either to upregulation of other Cav1.2 channels or to the existence of other mechanisms of insulin secretion unreliant on voltage gated channels as discussed next
Koike et al
Baseline MELD-3 AUC = 0.650) suggests that disease trajectory during treatment, rather than treatment itself, is the dominant factor determining outcomes
M.J.P.: R01DK106104 (NIH)
