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FEBS Lett 382: 223228
Cardiac iron overload in sickle-cell disease
The observed injuries in the lungs, heart, kidneys, and brain share a common mechanistic hallmark: glycocalyx shedding, increased vascular permeability, neutrophil adhesion, and oxidative stress [5, 17, 21, 104]
This viewpoint originated from observations using PET and later BOLD-fMRI, which frequently detect a larger increase in local CMRglc compared to the increase in CMRO during neural activation (Fox et al., 1988)
