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In DOX-treated cells and mouse hearts, levels of NLRP3 and related proteins were elevated, and calycosin (CA) ameliorated cardiotoxicity via the NLRP3 pathway (99)
Nonetheless, non-aggregated glycated forms are thought to be degraded by the proteasome, and their build-up can trigger ER stress 58,59
Bhatti JS, Bhatti GK, Reddy PH (2017) Mitochondrial dysfunction and oxidative stress in metabolic disordersa step towards mitochondria based therapeutic strategies
No therapy-combination recommendation should be made in public research-use content
