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IL-1-driven amyloid plaque clearance is associated with an expansion of transcriptionally reprogrammed microglia

Savvides, S

Similarly, exosomes from ASCs protect H9c2 cardiomyocytes from HO-induced damage by reducing apoptosis and hypertrophy in vitro 156

utilizing heteroatom zinc-philicity to induce uniform Zn 2+ nucleation, combined with high specific surface area to buffer volume variations, ultimately achieving synergistic optimization of energy density, power density, and cycling stability
