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Their multiple activities, including proliferation, differentiation, and ultimately cell death, are essential for ontogeny, homeostasis, and disease progression [1]
Microglia, the primary immune cells of the central nervous system, are deeply affected by signals originating from the gut microbiota, including microbial metabolites like short-chain fatty acids (SCFAs) and endotoxins such as lipopolysaccharides (LPS)
Additionally, the mechanism by which PNPLA3 inhibited lipolysis involved the re-esterification of mobilized FAs (Figure 1)
pylori by facilitating the in vivo attachment of H
