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Hyperosmolar stress stimulates generation of inflammatory cytokines, e.g., IL-1 and TNF-, promoting ocular surface inflammation [73]
In addition to activating the Akt signaling pathway, this drug facilitates apoptotic cell death, which is linked to increased phosphorylation of MEK and its downstream target ERK, decreased Bcl-2 levels, increased Bax expression, and greater activation of caspase-3 and caspase-9
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