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doi: 10.1016/j.intimp.2019.106044 135 ZhaoMHeHYinJ

Disease progression in a Transgenic model of Familial amyotrophic lateral sclerosis is dependent on both neuronal and non-neuronal zinc binding proteins

In PKD, besides induction of a maturation defect, lack of GSH in PK-deficient reticulocytes may further disrupt the already reduced PK-activity by oxidation of the enzyme, thereby initiating a deleterious downward spiral (van Berkel et al., 1973
