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However, GSH plays a major role in chemotherapy resistance as mentioned above, and inhibition of GSH as part of combined or prodrug therapies has been shown as a very effective approach for improving chemotherapies effectiveness
In ME/CFS, an increased prevalence of the class II major histocompatibility complex HLA-DQB*01 allele (88), along with two other variants of HLA-DQB1 in combination with two RAGE-374A variants (489) was found
Glutaredoxin-2 is required to control oxidative phosphorylation in cardiac muscle by mediating deglutathionylation reactions
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