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Description
Primary defects in carnitine synthesis or transport (deficiency OCTN2, trimethyllysine dioxygenase, butyrobetaine dioxygenase) are observed in about 1020% of ASD patients
These effects typically emerge during dose initiation or escalation and often diminish over time as the body adjusts to the medication
Nahrungsergnzungen sind kein Ersatz fr eine ausgewogene Ernhrung
Persistent regional changes in brain copper, cuproenzymes and catecholamines following perinatal copper deficiency in mice
