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Description
Targeting mitochondrial dysfunction and reactive oxygen species for neurodegenerative disease treatment
A possible explanation for the increased oxidative stress despite there being an increase in NADPH levels and no overall change in superoxide generation in this case is that G6PD-deficient myocardium are unable to fully alleviate oxidative stress
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Estimates suggest that the prevalence of DPN among adults with diabetes ranges from 6 to 51% [2, 3]
