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However, the current understanding of ferroptosis mechanisms and drug resistance in AML remains incomplete, and the targeting of ferroptosis, through the use of small molecule inhibitors, natural compounds and drug nano-administration, provides novel research directions and therapeutic possibilities for the treatment of AML
The SP1/ACSL4 pathway has been shown to be associated with ferroptosis and may serve as a potential therapeutic target in mouse models of intestinal ischemia necrosis and Alzheimer's disease [57, 58]
510, 497507.10.1002/cne.21808 49 GazzinS.ZelenkaJ.AdralovaL.KonickovaR.Coda ZabettaC.GiraudiP.BerengenoA
