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doi: 10.1152/ajprenal.00378.2021
Specific metabolic alterations in HCC involve the upregulation of FAO
Keser M
GDH1-derived AKG directly engages and activates the IKK-NF-B signaling axis, thereby upregulating the expression of the glucose transporter 1 (GLUT1) to promote compensatory glucose uptake and tumor cell survival under low glucose conditions [133]
