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CDK4 and CDK6 are important CDKs members that regulated the transition from G1 to S phase and bind to Cyclin Ds (D1, D2 and D3) to form CDK4/6-Cyclin complex after being activated by mitotic signals, while the over-activated CDK4/6 destabilizes the genome and chromosome, leading to uncontrolled proliferation and ultimately abnormal cell cycle regulation [44]

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doi: 10.3390/ijms19092812 15 CheesemanJ

Emerging evidence suggests that Nrf2 can enhance the expression of rhythm-inhibiting genes such as cryptochrome 2 and Rev-Erb in the nucleus, thereby indirectly inhibiting transcription mediated by the CLOCK-BMAL1 complex [103]