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However, the mechanisms of action for some inhibitors are not yet fully understood, and there are potential risks that remain unknown

The amplification of MUC1 gene led to over-expression of the MUC1 C-terminal subunit (MUC1-C) complex, which could mediate the TNBC cell self-renewal ability and tumor origin [373]
The elevated liver exosomal miR-2137 (Figure 6C) suggests that miR-2137 may be originally upregulated in the liver cells that have suffered from Cd exposure
Along with this, the activity of Cathepsin D and the protease is lower in colostrum than the milk (McGrath et al
