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The impact of ALA treatment on the various outcome measures monitored in the two studies indicates the magnitude of the contribution oxidative stress makes to the pathogenesis of glaucoma

doi: 10.2147/COPD.S10770

5A, the exposure of HUVECs to LP caused a dose-dependent increase in LKB1 (Ser428) phosphorylation, with a parallel increase in the phosphorylation of Thr172 of AMPK, a well-characterized enzyme downstream of LKB, in endothelial cells [59]
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