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Herein, we review the major metabolic pathways available to immune cells, then focus more closely on the role of FAO in different immune cell subsets

Crucially, genetic or pharmacological inhibition of AKT1 sensitized TNBC cells to cuproptosis, restoring DLAT lipoylation and oligomerization

Mast cells in general express H 1 and H 4 receptors, which have been implicated in the pathophysiology of peripheral type 1 hypersensitivity reactions and increased histamine and cytokine generation, respectively (Thangam et al., 2018) and guide chemotaxis (Hofstra et al., 2003

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