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The evidence indicates that acetylcarnitine may also play a beneficial role in the metabolism and functional recovery of aged hearts by increasing the acetylation of mitochondrial proteins [42]
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2021 Jun 9;2021:9949453
TMAO was found to exacerbate myocardial hypertrophy and cardiac fibrosis in rodent models with transverse aortic constriction and also in mouse models of doxorubicin (DOX)-induced cardiac fibrosis (Organ et al., 2016
