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The activation of clockophagy selectively degrades ARNTL through cargo receptor sequestosome 1 (SQSTM1), destabilizing HIF1A protein, and ultimately exacerbating lipid peroxidation and ferroptosis[71]
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Oxidative stress: adaptation, damage, repair and death
Additionally, GSTM1 null genotype is a risk factor for Alzheimers disease [67] and T-2 can suppress drug-metabolizing enzymes such as the GSTs [68] and related oxidative stress-associated pathways [68]
