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This reprogramming endows innate cells with a long-term, enhanced functional phenotype, enabling them to mount more robust and rapid pro-inflammatory, anti-infective and tissue repair responses against subsequent heterogeneous pathogenic challenges (Netea et al., 2016)

Activation of NOTCH signaling via DLL1 is mediated by APE1-redox-dependent NF-B activation in oesophageal adenocarcinoma

Ferroptosis, a regulated form of cell death triggered by iron-dependent lipid peroxidation and oxidative stress, has emerged as a potential targeted therapy for TNBC

Tacrine induces apoptosis through lysosome- and mitochondria-dependent pathway in HepG2 cells