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This subtype demonstrates elevated OXPHOS activity and increased mitochondrial gene expression, reflecting a reliance on mitochondrial metabolism rather than glycolysis for energy production (83)
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Hochster et al., 2014)
Cardiovascular effects beyond cardiac hypertrophy are poorly characterized for IGF-1 DES specifically, though IGF-1 influences cardiovascular physiology through effects on vascular smooth muscle, endothelial function, blood pressure regulation, and cardiac contractility - chronic perturbation of these processes carries unknown risks
