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Excess TG comes from hepatic fat production and dietary fat supply, while FFAs accumulate due to lipolysis of visceral adipose tissues (50)
The mechanism of action is that it affects the production of NAD+ on an intracellular level (Neelakantan, 2019)
GSH Metabolism GSH is abundant in the brain, with a high concentration in non-neuronal cells, mostly neuropil and white matter tracts, with the exception of some cerebellar neurons, such as granule cells and Purkinje cells [22]
